1.Neuronal caspase-3 signaling: not only cell death * M6 T* X' Y# v D5 t' N) V
3 R8 v' r$ G. T$ O7 M' \2.Apaf-1-independent programmed cell death in mouse development 7 O! C' \8 g o$ `' q 2 h* x0 w) I/ x5 ?3.Ezrin is a negative regulator of death receptor-induced apoptosis 9 D" }! @, h) R3 S2 N0 c' b" u
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4.Caspase 2-mediated tumor suppression involves survivin gene silencing 5 z, f+ m: ^7 E. B/ Y 4 d, J7 k6 k, G+ e7 I5.RIPK1 is not essential for TNFR1-induced activation of NF-κB & S& K1 N$ g! q( p" N % t5 w( }7 ^/ s& H5 z6.Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases 6 ~) o: {8 c0 M) P- j! E& o- ?/ q
, w _/ N \! ?) H7.Caspase-dependent generation of reactive oxygen species in human astrocytoma cells contributes to resistance to TRAIL-mediated apoptosis ( d: ] S6 e$ b, W# d 7 p& E* ]2 z. y3 w& l8.Radiation and anticancer drugs can facilitate mitochondrial bypass by CD95/Fas via c-FLIP downregulation 8 k% J/ |* C% A+ H2 J
9 I; A$ m4 G8 q+ o9.Caspase-mediated inhibition of sphingomyelin synthesis is involved in FasL-triggered cell death 9 f+ o2 L, [7 X& @
2 x, P( O3 ?2 G3 W10.Caspase-3-mediated secretion of connective tissue growth factor by apoptotic endothelial cells promotes fibrosis 2 \# E# R! P$ d7 I0 z3 a7 w* o; j" U& l
11.Restraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2 E1 t* H! D: ^4 S2 A
& ~( @9 h( x3 d12.CD95 engagement mediates actin-independent and -dependent apoptotic signals 7 V" I$ Y+ l3 B% K5 j2 g* f4 Y2 |: b# w% c: U2 c
13.Paclitaxel promotes a caspase 8-mediated apoptosis through death effector domain association with microtubules 2 w% j) b4 X# a2 t6 K f5 y' g( e3 y- g6 Z( ^3 x; u" I4 P
14.Apoptosis and glutathione: beyond an antioxidant 5 T" o7 [7 P4 O) w. v+ z
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15.BH3-only proteins in apoptosis and beyond: an overview 3 F9 S0 J6 m9 y - p" G, N( c2 N16.BH3-only proteins and their roles in programmed cell death 0 u8 W* }2 T0 h5 l9 y0 }6 {0 E* O) y2 N7 L, z; w5 Q6 O. U. x. F% [
17.Bid: a Bax-like BH3 protein ) l4 M: {- k0 R2 B
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18.Caspase-2: killer, savior and safeguard—emerging versatile roles for an ill-defined caspase ; k8 X6 {& P4 n, {' A0 h; S+ ?; P
( G6 F! ^* A0 Y8 n* X19.Beclin 1: a BH3-only protein that fails to induce apoptosis # D5 p" L6 D6 f9 J! V; | ) J+ W5 m# C! ~' p/ c( H/ f9 C20.DISC-mediated activation of caspase-2 in DNA damage-induced apoptosis ; }) f' E+ u3 K; \+ ^* b
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21.Bcl-2 complexed with Beclin-1 maintains full anti-apoptotic function 0 m$ ^5 u3 b" {' \& S: q5 b' I& W; |2 C1 O' O" m
22.Far upstream element-binding protein-1, a novel caspase substrate, acts as a cross-talker between apoptosis and the c-myc oncogene & v7 v7 p; v( q% Y4 P' T# ~ , u, U: J9 D0 I# D/ R" ^7 t23.Apoptotic signaling by c-MYC 1 x) h! k* x% T: l& @) L5 H
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24.Caspase-independent cell death: leaving the set without the final cut - H6 E: S0 j& d* s8 u [0 k, } Z7 M
25.Caspases in apoptosis and beyond作者: ji_qing1 时间: 2010-11-18 15:13
