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False-colored scanning electron micrograph of erythrocytes/ X! Z7 r6 ^, u5 W) B7 y
(diameter ~7 micrometers). Sickle cell disease results from an
5 Z- r' } ?( C7 N s9 ninherited defect of adult hemoglobin, the oxygen-carrying
; N ^# t. U* F3 C5 xmetalloprotein constituent of erythrocytes. Common genetic variation
$ ?: w" j# L! _7 v$ Tassociated with sickle cell disease severity modulates an adult-stage
( |/ h: t/ t9 `" s" Serythroid enhancer element of the BCL11A gene. Disruption of this# X) ?+ Q7 }* [, G" C5 u; |0 i; u X
element could ameliorate the disease by reestablishing expression
" V* q. \1 z; rof fetal hemoglobin. See pages 206 and 253.
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?. X/ J$ }. D% i3 z, }5 z: n0 S3 P d: ~! `
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发表于 2013-10-12 08:20
发表于 2013-10-12 08:41
