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False-colored scanning electron micrograph of erythrocytes
4 h8 v/ Q3 w/ b9 ?(diameter ~7 micrometers). Sickle cell disease results from an
& `' u8 }1 U& ^0 T/ Dinherited defect of adult hemoglobin, the oxygen-carrying
2 {$ H6 X+ e3 `$ W4 {# |1 ametalloprotein constituent of erythrocytes. Common genetic variation) ]1 Q E# p, u* |9 X7 T/ X4 T
associated with sickle cell disease severity modulates an adult-stage
$ n' Y0 g3 n" j: q; lerythroid enhancer element of the BCL11A gene. Disruption of this
6 S, N- l1 A8 G; uelement could ameliorate the disease by reestablishing expression& w1 X# W) d1 F/ D+ O6 i* T
of fetal hemoglobin. See pages 206 and 253.
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发表于 2013-10-12 08:20
发表于 2013-10-12 08:41
