文献求助

yuhaoze

1.Neuronal caspase-3 signaling: not only cell death & P4 y: p) S$ P' ]/ J3 q: M8 E
4 A& i" R/ z) ]7 T
2.Apaf-1-independent programmed cell death in mouse development
6 R- A+ `, I! i3 Z( t" t+ |; K/ O& n/ |/ `2 o& S
3.Ezrin is a negative regulator of death receptor-induced apoptosis
* D+ I- N8 ]& j* K0 H! l% y, I4 N( }% D# b/ k% p% f
4.Caspase 2-mediated tumor suppression involves survivin gene silencing
" K8 M# o: G# f4 V8 b- @) d) c( ?1 P' k% {7 @) t$ y0 b
5.RIPK1 is not essential for TNFR1-induced activation of NF-κB 3 k$ V2 V+ E/ O/ f9 H5 }) c/ m
4 i8 A* L2 u+ ]8 q  M5 z
6.Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases
! [& V+ k4 a0 Q. D' Y( K
% `( M% Q( x8 o6 `6 h" [. E1 B: Q7.Caspase-dependent generation of reactive oxygen species in human astrocytoma cells contributes to resistance to TRAIL-mediated apoptosis
7 O, Q0 Q) N. C% }8 {( ?$ N, `3 x) {, y7 z
8.Radiation and anticancer drugs can facilitate mitochondrial bypass by CD95/Fas via c-FLIP downregulation % C1 a  e  F, M

& b, c+ o; T) f- z9.Caspase-mediated inhibition of sphingomyelin synthesis is involved in FasL-triggered cell death 2 z+ ]7 o  x% A' k  ?" s, R

2 b9 Q% \; F8 w10.Caspase-3-mediated secretion of connective tissue growth factor by apoptotic endothelial cells promotes fibrosis ( j- P3 F& ~8 W4 E. f1 o# @
( p# i5 w# _& K. _$ r3 D% t
11.Restraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2
' @) O5 r: \. t: ?; K
+ X; k" y6 E6 w1 y- G' B12.CD95 engagement mediates actin-independent and -dependent apoptotic signals
. O. \  g% k* _9 b( z0 V5 F0 N, J6 ^# @7 J( v
13.Paclitaxel promotes a caspase 8-mediated apoptosis through death effector domain association with microtubules
2 }+ i. ~/ }* L1 c* ?; l0 ^4 _+ s, ]* g1 [: P8 `, {
14.Apoptosis and glutathione: beyond an antioxidant
) E3 ?' e+ J$ F9 ~( ^$ \
4 g9 \+ h/ _3 Q$ e15.BH3-only proteins in apoptosis and beyond: an overview
/ h& e' O- {- q4 o; c9 g9 t( p3 N2 V: @" r  U
16.BH3-only proteins and their roles in programmed cell death 4 e& a# X: P' |  Q
! c; D  L: H2 N9 J0 }2 W% j, E
17.Bid: a Bax-like BH3 protein
8 Q' e0 d% d6 a9 A: u1 N+ v4 |( x2 q' k2 X- S5 t5 ~
18.Caspase-2: killer, savior and safeguard—emerging versatile roles for an ill-defined caspase
- ~6 P3 k, ]8 u/ [+ G8 e$ G/ ^. {* n; E
19.Beclin 1: a BH3-only protein that fails to induce apoptosis
6 }. o6 _/ i! w+ Q, e- y" p, o# t7 @' b5 u; v; \
20.DISC-mediated activation of caspase-2 in DNA damage-induced apoptosis 6 f7 |9 R% z3 O% Y' s9 `4 r/ h
+ z  ^0 F) T% W* x# G* h( w$ s
21.Bcl-2 complexed with Beclin-1 maintains full anti-apoptotic function ; b- k6 Q6 m  b1 o' _- K

( e, @9 t  Z7 [. |) _22.Far upstream element-binding protein-1, a novel caspase substrate, acts as a cross-talker between apoptosis and the c-myc oncogene
  b# S7 q6 _/ ]. z3 q& Q% ^; f0 K6 P0 B. s3 U: c
23.Apoptotic signaling by c-MYC
  p4 i* g/ f, B1 h: S; i
3 I: N& ]0 w: x; H( E! c+ A24.Caspase-independent cell death: leaving the set without the final cut
6 s. [8 }& C9 f* ~) J/ y8 x
- K( z" D& \( {  r25.Caspases in apoptosis and beyond

ssuwhite

回复 1# yuhaoze

yuhaoze

回复 10# hstudent
: ^! X6 u: [( g* U0 S$ r
' z0 [- U$ v4 ^$ t6 [  [6 J2 ^, c3 E! Z; Q- R
    谢谢你！！

hstudent

11 偶看ok
2 i" x. T- h8 V3 y# G. ?2 rRestraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2

hstudent

14.偶看ok2 ^' s0 C- o5 E9 V7 |( J, Q3 {: z( d

! D: F1 b7 Z' e  v. vApoptosis and glutathione: beyond an antioxidant

hstudent

17 偶看ok1 ^/ W: D5 X0 q' v! z

- r1 D5 b0 K1 `3 f( k1 D/ ABid: a Bax-like BH3 protein

hstudent

5.ok ok
: T+ J+ F1 R7 V; k) [RIPK1 is not essential for TNFR1-induced activation of NF-κB

hstudent

2. ok ok
6 u' c2 k! L) ~& \Apaf-1-independent programmed cell death in mouse development

yuhaoze

回复 3# 细胞海洋 6 s' F3 ~$ P3 P# e4 T% v+ |

& U1 T2 A& o: m) W! M3 R9 n: t) x5 l$ _1 z2 V" }. ?( [1 [
    知道啦

yuhaoze

回复 2# ji_qing1
3 O8 {0 t! y' s% K; g
5 J" [0 z; b; e2 Q; a1 K/ H7 B- b
$ k. D% v' \' L8 y- \4 {/ a$ f    哪，我想看的最近几年的oncogene都下不到