|
  
- 积分
- 1354
- 威望
- 1354
- 包包
- 484
|

1.Neuronal caspase-3 signaling: not only cell death
% b% g1 j; x0 V$ Q& u0 J: V5 q8 Z8 Q
0 ~/ i1 Q7 p6 k d2.Apaf-1-independent programmed cell death in mouse development + J+ i4 M1 j. J7 @$ q
7 }% Q6 Y9 O0 B" `( N& @1 O6 }3.Ezrin is a negative regulator of death receptor-induced apoptosis
* t3 A s; \. m- M# ]# S0 g j9 f& }4 X
4.Caspase 2-mediated tumor suppression involves survivin gene silencing : I2 I( O2 m- X$ |/ h
9 _$ L' p$ G D* {- h. I( i
5.RIPK1 is not essential for TNFR1-induced activation of NF-κB 3 p- J$ _- l& B
% ~. B: \& r+ c6 n3 l- \ N0 h s6.Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases * R" O4 a5 I0 n# b5 M, F; B2 V
0 P- `# C1 L! W/ Q6 i+ T- Z) X7.Caspase-dependent generation of reactive oxygen species in human astrocytoma cells contributes to resistance to TRAIL-mediated apoptosis
1 [" S0 F" o# O$ H$ F4 @8 Q; R5 z, v! p e( _( d( R# R
8.Radiation and anticancer drugs can facilitate mitochondrial bypass by CD95/Fas via c-FLIP downregulation " T1 i7 U \5 G
5 @" b6 I( _2 t9.Caspase-mediated inhibition of sphingomyelin synthesis is involved in FasL-triggered cell death
! @8 T. \! J# }" ]3 I$ R# j- W% B5 `- k% j5 _) A
10.Caspase-3-mediated secretion of connective tissue growth factor by apoptotic endothelial cells promotes fibrosis
* g% y& C, T7 _" Z! ]
% y) a( }4 y3 l11.Restraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2
, ^ I$ K0 T( e/ M. f* _7 a- \
& k+ ]5 a+ g/ C" N12.CD95 engagement mediates actin-independent and -dependent apoptotic signals $ G: p$ @0 p1 ?7 V, m8 s4 m
# T5 l, G4 e: g7 c13.Paclitaxel promotes a caspase 8-mediated apoptosis through death effector domain association with microtubules
$ h" U# B# R, G6 E! c7 @: M/ o1 p2 }' H9 R- y3 ^
14.Apoptosis and glutathione: beyond an antioxidant
6 ]' ^4 Y0 Y: ]9 p/ s
! I+ c8 S4 K. y& b15.BH3-only proteins in apoptosis and beyond: an overview
2 P) f: ^" F7 S( k G1 d. c$ ^
5 \5 x4 c8 g l( W8 I! S16.BH3-only proteins and their roles in programmed cell death p4 q: l+ A3 P" S7 G8 O
( A/ h) h+ w& l. \$ E+ _; s
17.Bid: a Bax-like BH3 protein ; ]9 B6 s4 p& i& Q/ I
6 q& c6 F- h. Q+ u" l: x8 v+ y18.Caspase-2: killer, savior and safeguard—emerging versatile roles for an ill-defined caspase 7 _8 e5 h# f+ q9 |; |* q4 O
1 ?2 Y' q' ?, m+ _
19.Beclin 1: a BH3-only protein that fails to induce apoptosis * d" M. C5 ?* e+ A
* N+ {5 u) S. U
20.DISC-mediated activation of caspase-2 in DNA damage-induced apoptosis
4 D; r# w6 T0 o5 p# g; D& ]* x$ k3 |: C0 ^7 O8 i7 O) t3 v( M: J
21.Bcl-2 complexed with Beclin-1 maintains full anti-apoptotic function
- X+ M3 _5 P. b# g! a' P. l+ E3 ~% b/ i* e
22.Far upstream element-binding protein-1, a novel caspase substrate, acts as a cross-talker between apoptosis and the c-myc oncogene
- H" d7 u. O8 w* F
: ?3 R/ m. e" o/ x# j% I5 N23.Apoptotic signaling by c-MYC
, z2 ]1 b* B2 @5 \; l& ?9 r* v7 X2 ^: G
24.Caspase-independent cell death: leaving the set without the final cut
2 |- P- Y: G/ P: N+ N% H2 D5 A0 |# w# d4 b5 q% s3 l
25.Caspases in apoptosis and beyond |
|