文献求助

yuhaoze

1.Neuronal caspase-3 signaling: not only cell death
. R2 y. {7 d8 S8 o$ r2 t. H8 L; o# T7 A; m0 f0 X
2.Apaf-1-independent programmed cell death in mouse development 2 s' B; E8 ^; h. j$ N
/ i" R2 s  R/ Z, h: h1 y( t' P
3.Ezrin is a negative regulator of death receptor-induced apoptosis ! E  |" X! z6 M. J
# L+ |1 z9 {4 B4 g1 A" ^
4.Caspase 2-mediated tumor suppression involves survivin gene silencing ! Z9 ?, F  ~  D8 C7 Q$ g0 Y& N
0 |0 a' p( ?/ ?! Q. T
5.RIPK1 is not essential for TNFR1-induced activation of NF-κB + O7 C9 K& A* @' w# T
+ T7 J& Q$ E8 F& Q/ c/ }
6.Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases ; H5 Q5 ?  c- D( }

. H; E( x# ~! E( c$ I* X7.Caspase-dependent generation of reactive oxygen species in human astrocytoma cells contributes to resistance to TRAIL-mediated apoptosis / T6 t7 V& J! d* h

+ x' P8 k9 _) v3 n  t+ z" O4 O$ h8.Radiation and anticancer drugs can facilitate mitochondrial bypass by CD95/Fas via c-FLIP downregulation
0 V8 z4 o3 T3 ~4 A1 {: D$ ^( X
9.Caspase-mediated inhibition of sphingomyelin synthesis is involved in FasL-triggered cell death
4 q4 `* V+ B, B: ?6 e) q. U6 F1 J! m' W
10.Caspase-3-mediated secretion of connective tissue growth factor by apoptotic endothelial cells promotes fibrosis : L2 M+ G- A- w; q! r/ k* h

8 F- q) O& L% _11.Restraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2$ g* X! J+ D# Y' h

7 }6 v% E" A; i( |$ u) V4 z12.CD95 engagement mediates actin-independent and -dependent apoptotic signals
; G: a1 r4 k& T# P2 [
( F: ]! v5 c5 G: D% s* m  Z13.Paclitaxel promotes a caspase 8-mediated apoptosis through death effector domain association with microtubules
: B- U& r* Z- |0 W  ^8 a' p% u. N2 S1 T
14.Apoptosis and glutathione: beyond an antioxidant 7 u6 L4 U7 T4 l" t) ?

1 N  {/ v* X1 ~& m0 Q/ U* A  c15.BH3-only proteins in apoptosis and beyond: an overview
/ ]8 c0 W# i( E+ b  Y
9 l3 x$ v2 ~0 @+ @) e16.BH3-only proteins and their roles in programmed cell death
2 K) s6 b6 }4 t7 B/ X* z$ a6 l
; h( l/ Y1 Y7 I& _  Z" B17.Bid: a Bax-like BH3 protein 5 n' Y2 ?& I+ g1 Z) [
/ ~! f: ^" f$ n: v- e
18.Caspase-2: killer, savior and safeguard—emerging versatile roles for an ill-defined caspase 5 G3 p& p- l$ Y7 X( g) T& [, n2 r
7 ~7 @8 P) A7 a) M
19.Beclin 1: a BH3-only protein that fails to induce apoptosis
' J* {' P8 o2 y  q  d0 r% q7 d6 q8 h; N4 P" O4 }- b
20.DISC-mediated activation of caspase-2 in DNA damage-induced apoptosis - |: l) y& ]5 G8 J
- P$ h6 r4 A# u0 t+ I
21.Bcl-2 complexed with Beclin-1 maintains full anti-apoptotic function 5 p/ Z. n4 f% R6 x( ~& j

; V  q9 V- \# o- h22.Far upstream element-binding protein-1, a novel caspase substrate, acts as a cross-talker between apoptosis and the c-myc oncogene 4 v( R% g5 Y, R6 u6 d

# M. d& z5 P9 T/ Z7 X$ p; x23.Apoptotic signaling by c-MYC 4 |$ G; H8 t3 i% O6 N  `4 X
' i! i  p5 g' k- P% G
24.Caspase-independent cell death: leaving the set without the final cut & }- r/ d  ~6 \1 h* B1 O5 R) L
+ I1 t- k7 m; W9 G( v
25.Caspases in apoptosis and beyond

yuhaoze

回复 2# ji_qing1 & D9 T# j% v+ o! Y, O& Q) F

6 ~: |- G7 d5 p: Y- E$ h. {
! ~% H1 r8 i3 b; I    哪，我想看的最近几年的oncogene都下不到

yuhaoze

回复 3# 细胞海洋
9 D& G, [5 R+ H! \- V
) V1 _( t8 w8 C4 }' z# x* k2 M- o7 Q& f  N+ }4 b/ f
    知道啦

yuhaoze

回复 10# hstudent 2 u6 s; P/ e1 S) C

/ p7 ]/ G9 m" P8 D' r4 K# w5 ^! P2 ~! v1 D
    谢谢你！！