  
- 积分
- 1354
- 威望
- 1354
- 包包
- 484
|
1.Neuronal caspase-3 signaling: not only cell death
4 {, l9 h+ x. J7 y& j% z3 k5 y+ @# R" u8 D _: [+ L9 z
2.Apaf-1-independent programmed cell death in mouse development 2 q5 H3 o: Y7 T
9 F; e" Y3 P$ a; g# b
3.Ezrin is a negative regulator of death receptor-induced apoptosis & g- z1 O0 p' F2 \
$ ?, P; ~5 J5 ^ ^
4.Caspase 2-mediated tumor suppression involves survivin gene silencing
! H0 \- S8 x" I5 i( i$ U
2 ~9 ?9 y0 \' F, o0 x$ m5.RIPK1 is not essential for TNFR1-induced activation of NF-κB " V% q1 l) l# p; O; F- p
. c! m2 E/ s& c" q" x
6.Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases * w" ?* u* ]9 D9 i& o/ A" O7 V
# ^$ F: U3 T4 V0 L Y4 |7.Caspase-dependent generation of reactive oxygen species in human astrocytoma cells contributes to resistance to TRAIL-mediated apoptosis 9 r) P- W. m; n3 U
& j$ C+ G; O* |' P4 ~
8.Radiation and anticancer drugs can facilitate mitochondrial bypass by CD95/Fas via c-FLIP downregulation * ]# T8 Z, G* x
7 L3 L' L+ l; r9.Caspase-mediated inhibition of sphingomyelin synthesis is involved in FasL-triggered cell death
Z- J9 F- U$ t8 O1 t; g) @
* X0 F' j3 s0 i" {+ `: L' ~10.Caspase-3-mediated secretion of connective tissue growth factor by apoptotic endothelial cells promotes fibrosis ) |, {, S5 v6 U! T5 J! I3 `/ e
$ d$ W' K, ^1 I1 k# a11.Restraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2
$ Z0 I* h7 x2 Y+ t& W
$ |, j, l! e. }2 Y& y" t, x# _12.CD95 engagement mediates actin-independent and -dependent apoptotic signals 2 I, }% K y' X% B; p( y
% j. y' t9 q$ `3 X( J13.Paclitaxel promotes a caspase 8-mediated apoptosis through death effector domain association with microtubules , A$ `. c h2 E g
" l# u S4 |8 n0 i7 \14.Apoptosis and glutathione: beyond an antioxidant
" `4 L2 A3 ~6 Q& M6 \( a1 q& s" o k. x* I
15.BH3-only proteins in apoptosis and beyond: an overview ( K2 w9 r: ?. B3 N& M# L* ~
5 \1 Z" z, @: @- e
16.BH3-only proteins and their roles in programmed cell death
9 Y/ I' B0 k! y; I) P1 [ Y
2 G1 Y) s( L. n; U0 C17.Bid: a Bax-like BH3 protein ; z& H% g. b( A+ h$ S6 z7 B
. u* w+ v E+ I+ v18.Caspase-2: killer, savior and safeguard—emerging versatile roles for an ill-defined caspase ( f. z2 q% [1 v
4 {. T z6 ?0 D0 N- g
19.Beclin 1: a BH3-only protein that fails to induce apoptosis
, f% M7 w& g( R i6 u
* K7 J# o. K/ V0 ^& K6 \20.DISC-mediated activation of caspase-2 in DNA damage-induced apoptosis 0 ~. n4 U5 w9 s) G9 P
3 G6 i. {4 C( G7 E21.Bcl-2 complexed with Beclin-1 maintains full anti-apoptotic function
: W. \' n# o! t4 o |+ k- p# E, ]
22.Far upstream element-binding protein-1, a novel caspase substrate, acts as a cross-talker between apoptosis and the c-myc oncogene
- [9 T0 z H2 T* K" a: I
9 K7 `- f0 h/ g: }$ B23.Apoptotic signaling by c-MYC 3 y0 B/ Z8 A# {3 Z0 ?- ]% Z
6 C% e. [5 R/ Q# `' @
24.Caspase-independent cell death: leaving the set without the final cut ' K! r5 E6 n# d; y4 V7 d
. T, m' U* ~ z) b3 y25.Caspases in apoptosis and beyond |
|